Betanin Promotes Wound Closure and Drives a Context-Specific Transcriptional Repair Program in HaCaT Keratinocytes: A Multi-Evidence AI-Guided Prioritization Study.
- DOI
- 10.1002/biof.70151
- Published
- 2026 Sep-Oct
- Container
- BioFactors (Oxford, England)
- Publisher
- Not recorded
- Open access
- yes
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limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.
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Cite this work
BibTeX
@article{allodium:10.1002/biof.70151,
title = {Betanin Promotes Wound Closure and Drives a Context-Specific Transcriptional Repair Program in HaCaT Keratinocytes: A Multi-Evidence AI-Guided Prioritization Study.},
author = {Park SY and Kim Y},
year = {2026},
journal = {BioFactors (Oxford, England)},
doi = {10.1002/biof.70151},
url = {https://doi.org/10.1002/biof.70151}
}RIS
TY - JOUR TI - Betanin Promotes Wound Closure and Drives a Context-Specific Transcriptional Repair Program in HaCaT Keratinocytes: A Multi-Evidence AI-Guided Prioritization Study. AU - Park SY AU - Kim Y PY - 2026 JO - BioFactors (Oxford, England) DO - 10.1002/biof.70151 UR - https://doi.org/10.1002/biof.70151 ER -
APA
SY, P., & Y, K. (2026). Betanin Promotes Wound Closure and Drives a Context-Specific Transcriptional Repair Program in HaCaT Keratinocytes: A Multi-Evidence AI-Guided Prioritization Study.. BioFactors (Oxford, England). https://doi.org/10.1002/biof.70151
Source records
- pubmed · retrieved 2026-09-24T22:31:39.799Z