Selective AUTAC-mediated ATP7B degradation unlocks cuproptosis vulnerability in pancreatic ductal adenocarcinoma.

Zhang Z, Li M, Feng J, Li JA, Zhang L

DOI: 10.1002/ctm2.70848
Published: 2026 Nov
Container: Clinical and translational medicine
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.1002/ctm2.70848,
  title = {Selective AUTAC-mediated ATP7B degradation unlocks cuproptosis vulnerability in pancreatic ductal adenocarcinoma.},
  author = {Zhang Z and Li M and Feng J and Li JA and Zhang L},
  year = {2026},
  journal = {Clinical and translational medicine},
  doi = {10.1002/ctm2.70848},
  url = {https://doi.org/10.1002/ctm2.70848}
}

RIS

TY  - JOUR
TI  - Selective AUTAC-mediated ATP7B degradation unlocks cuproptosis vulnerability in pancreatic ductal adenocarcinoma.
AU  - Zhang Z
AU  - Li M
AU  - Feng J
AU  - Li JA
AU  - Zhang L
PY  - 2026
JO  - Clinical and translational medicine
DO  - 10.1002/ctm2.70848
UR  - https://doi.org/10.1002/ctm2.70848
ER  -

APA

Z, Z., M, L., J, F., JA, L., & L, Z. (2026). Selective AUTAC-mediated ATP7B degradation unlocks cuproptosis vulnerability in pancreatic ductal adenocarcinoma.. Clinical and translational medicine. https://doi.org/10.1002/ctm2.70848

Source records

pubmed · retrieved 2026-11-10T06:51:41.576Z