Human mitochondria-derivedN-formylated peptides are novel agonists equally active on FPR and FPRL1, whileListeria monocytogenes-derived peptides preferentially activate FPR

Marie-Josèphe Rabiet, Emilie Huet, François Boulay

Open source

DOI: 10.1002/eji.200526338
Published: 2005-08
Container: European Journal of Immunology
Publisher: Wiley
Open access: unknown

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Cite this work

BibTeX

@article{allodium:10.1002/eji.200526338,
  title = {Human mitochondria-derivedN-formylated peptides are novel agonists equally active on FPR and FPRL1, whileListeria monocytogenes-derived peptides preferentially activate FPR},
  author = {Marie-Josèphe Rabiet and Emilie Huet and François Boulay},
  year = {2005},
  journal = {European Journal of Immunology},
  doi = {10.1002/eji.200526338},
  url = {https://doi.org/10.1002/eji.200526338}
}

RIS

TY  - JOUR
TI  - Human mitochondria-derivedN-formylated peptides are novel agonists equally active on FPR and FPRL1, whileListeria monocytogenes-derived peptides preferentially activate FPR
AU  - Marie-Josèphe Rabiet
AU  - Emilie Huet
AU  - François Boulay
PY  - 2005
JO  - European Journal of Immunology
DO  - 10.1002/eji.200526338
UR  - https://doi.org/10.1002/eji.200526338
ER  -

APA

Rabiet, M., Huet, E., & Boulay, F. (2005). Human mitochondria-derivedN-formylated peptides are novel agonists equally active on FPR and FPRL1, whileListeria monocytogenes-derived peptides preferentially activate FPR. European Journal of Immunology. https://doi.org/10.1002/eji.200526338

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crossref · retrieved 2026-11-10T18:09:46.256Z