Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer Formation.

Hagl B, Spielberger BD, Neumann B, Pelham SJ, Pandey D, Schlundt A, Barro C, Lechner A, Wolf C, Deenick EK, Sattler M, Tangye SG, Rothenfusser S, Renner ED

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DOI: 10.1002/eji.70015
Published: 2025 Jul
Container: European journal of immunology
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.1002/eji.70015,
  title = {Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer Formation.},
  author = {Hagl B and Spielberger BD and Neumann B and Pelham SJ and Pandey D and Schlundt A and Barro C and Lechner A and Wolf C and Deenick EK and Sattler M and Tangye SG and Rothenfusser S and Renner ED},
  year = {2025},
  journal = {European journal of immunology},
  doi = {10.1002/eji.70015},
  url = {https://doi.org/10.1002/eji.70015}
}

RIS

TY  - JOUR
TI  - Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer Formation.
AU  - Hagl B
AU  - Spielberger BD
AU  - Neumann B
AU  - Pelham SJ
AU  - Pandey D
AU  - Schlundt A
AU  - Barro C
AU  - Lechner A
AU  - Wolf C
AU  - Deenick EK
AU  - Sattler M
AU  - Tangye SG
AU  - Rothenfusser S
AU  - Renner ED
PY  - 2025
JO  - European journal of immunology
DO  - 10.1002/eji.70015
UR  - https://doi.org/10.1002/eji.70015
ER  -

APA

B, H., BD, S., B, N., SJ, P., D, P., A, S., C, B., A, L., C, W., EK, D., M, S., SG, T., S, R., & ED, R. (2025). Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer Formation.. European journal of immunology. https://doi.org/10.1002/eji.70015

Source records

pubmed · retrieved 2026-09-25T22:41:59.079Z