TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity
- DOI
- 10.1007/s00204-016-1830-8
- Published
- 2016-09-01
- Container
- Archives of Toxicology
- Publisher
- Springer Science and Business Media LLC
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1007/s00204-016-1830-8,
title = {TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity},
author = {Cristian González-Guerrero and Pablo Cannata-Ortiz and Consuelo Guerri and Jesús Egido and Alberto Ortiz and Adrián M. Ramos},
year = {2016},
journal = {Archives of Toxicology},
doi = {10.1007/s00204-016-1830-8},
url = {https://doi.org/10.1007/s00204-016-1830-8}
}RIS
TY - JOUR TI - TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity AU - Cristian González-Guerrero AU - Pablo Cannata-Ortiz AU - Consuelo Guerri AU - Jesús Egido AU - Alberto Ortiz AU - Adrián M. Ramos PY - 2016 JO - Archives of Toxicology DO - 10.1007/s00204-016-1830-8 UR - https://doi.org/10.1007/s00204-016-1830-8 ER -
APA
González-Guerrero, C., Cannata-Ortiz, P., Guerri, C., Egido, J., Ortiz, A., & Ramos, A. M. (2016). TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity. Archives of Toxicology. https://doi.org/10.1007/s00204-016-1830-8
Source records
- crossref · retrieved 2026-09-27T16:18:18.031Z