DOT1L-mediated H3K79me3 of ITCH promotes AURKA ubiquitination to suppress ECM degradation in osteoarthritis

Duo Xia, Jialiang Li, Xuanping Zhou, Chun Yu, Shu Yang

Open source

DOI: 10.1007/s00438-026-02514-z
Published: 2026-09-09
Container: Molecular Genetics and Genomics
Publisher: Springer Science and Business Media LLC
Open access: unknown

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Cite this work

BibTeX

@article{allodium:10.1007/s00438-026-02514-z,
  title = {DOT1L-mediated H3K79me3 of ITCH promotes AURKA ubiquitination to suppress ECM degradation in osteoarthritis},
  author = {Duo Xia and Jialiang Li and Xuanping Zhou and Chun Yu and Shu Yang},
  year = {2026},
  journal = {Molecular Genetics and Genomics},
  doi = {10.1007/s00438-026-02514-z},
  url = {https://doi.org/10.1007/s00438-026-02514-z}
}

RIS

TY  - JOUR
TI  - DOT1L-mediated H3K79me3 of ITCH promotes AURKA ubiquitination to suppress ECM degradation in osteoarthritis
AU  - Duo Xia
AU  - Jialiang Li
AU  - Xuanping Zhou
AU  - Chun Yu
AU  - Shu Yang
PY  - 2026
JO  - Molecular Genetics and Genomics
DO  - 10.1007/s00438-026-02514-z
UR  - https://doi.org/10.1007/s00438-026-02514-z
ER  -

APA

Xia, D., Li, J., Zhou, X., Yu, C., & Yang, S. (2026). DOT1L-mediated H3K79me3 of ITCH promotes AURKA ubiquitination to suppress ECM degradation in osteoarthritis. Molecular Genetics and Genomics. https://doi.org/10.1007/s00438-026-02514-z

Source records

crossref · retrieved 2026-09-25T20:46:01.294Z