Angelica sinensis polysaccharide reduces macrophage FPR2 to regulate the hepatic inflammatory microenvironment and activate the PI3K/AKT signaling pathway to improve insulin resistance.
- DOI
- 10.1007/s10616-026-01071-y
- Published
- 2026 Oct
- Container
- Cytotechnology
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1007/s10616-026-01071-y,
title = {Angelica sinensis polysaccharide reduces macrophage FPR2 to regulate the hepatic inflammatory microenvironment and activate the PI3K/AKT signaling pathway to improve insulin resistance.},
author = {Duan Z and Peng J and Zhu H and Ruan B and Cao M},
year = {2026},
journal = {Cytotechnology},
doi = {10.1007/s10616-026-01071-y},
url = {https://doi.org/10.1007/s10616-026-01071-y}
}RIS
TY - JOUR TI - Angelica sinensis polysaccharide reduces macrophage FPR2 to regulate the hepatic inflammatory microenvironment and activate the PI3K/AKT signaling pathway to improve insulin resistance. AU - Duan Z AU - Peng J AU - Zhu H AU - Ruan B AU - Cao M PY - 2026 JO - Cytotechnology DO - 10.1007/s10616-026-01071-y UR - https://doi.org/10.1007/s10616-026-01071-y ER -
APA
Z, D., J, P., H, Z., B, R., & M, C. (2026). Angelica sinensis polysaccharide reduces macrophage FPR2 to regulate the hepatic inflammatory microenvironment and activate the PI3K/AKT signaling pathway to improve insulin resistance.. Cytotechnology. https://doi.org/10.1007/s10616-026-01071-y
Source records
- pubmed · retrieved 2026-09-25T21:59:42.694Z