MEK/ERK pathway activation by insulin receptor isoform alteration is associated with the abnormal proliferation and differentiation of intestinal epithelial cells in diabetic mice.

Ouyang H, Yang HS, Yu T, Shan TD, Li JY, Huang CZ, Zhong W, Xia ZS, Chen QK

Open source

DOI
10.1007/s11010-015-2650-5
Published
2016 Feb
Container
Molecular and cellular biochemistry
Publisher
Not recorded
Open access
unknown

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BibTeX

@article{allodium:10.1007/s11010-015-2650-5,
  title = {MEK/ERK pathway activation by insulin receptor isoform alteration is associated with the abnormal proliferation and differentiation of intestinal epithelial cells in diabetic mice.},
  author = {Ouyang H and Yang HS and Yu T and Shan TD and Li JY and Huang CZ and Zhong W and Xia ZS and Chen QK},
  year = {2016},
  journal = {Molecular and cellular biochemistry},
  doi = {10.1007/s11010-015-2650-5},
  url = {https://doi.org/10.1007/s11010-015-2650-5}
}

RIS

TY  - JOUR
TI  - MEK/ERK pathway activation by insulin receptor isoform alteration is associated with the abnormal proliferation and differentiation of intestinal epithelial cells in diabetic mice.
AU  - Ouyang H
AU  - Yang HS
AU  - Yu T
AU  - Shan TD
AU  - Li JY
AU  - Huang CZ
AU  - Zhong W
AU  - Xia ZS
AU  - Chen QK
PY  - 2016
JO  - Molecular and cellular biochemistry
DO  - 10.1007/s11010-015-2650-5
UR  - https://doi.org/10.1007/s11010-015-2650-5
ER  - 

APA

H, O., HS, Y., T, Y., TD, S., JY, L., CZ, H., W, Z., ZS, X., & QK, C. (2016). MEK/ERK pathway activation by insulin receptor isoform alteration is associated with the abnormal proliferation and differentiation of intestinal epithelial cells in diabetic mice.. Molecular and cellular biochemistry. https://doi.org/10.1007/s11010-015-2650-5

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