18β-glycyrrhetinic acid ameliorates ifosfamide-induced nephrotoxicity by suppressing Wnt3a/IL-17A-driven inflammatory signaling, ER stress, and apoptosis while preserving ZO-1 integrity.
- DOI
- 10.1007/s11010-026-05748-y
- Published
- 2026 Sep 21
- Container
- Molecular and cellular biochemistry
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1007/s11010-026-05748-y,
title = {18β-glycyrrhetinic acid ameliorates ifosfamide-induced nephrotoxicity by suppressing Wnt3a/IL-17A-driven inflammatory signaling, ER stress, and apoptosis while preserving ZO-1 integrity.},
author = {Kandemir FM and Şimşek H and Çelik Samancı T and Kandemir Ö},
year = {2026},
journal = {Molecular and cellular biochemistry},
doi = {10.1007/s11010-026-05748-y},
url = {https://doi.org/10.1007/s11010-026-05748-y}
}RIS
TY - JOUR TI - 18β-glycyrrhetinic acid ameliorates ifosfamide-induced nephrotoxicity by suppressing Wnt3a/IL-17A-driven inflammatory signaling, ER stress, and apoptosis while preserving ZO-1 integrity. AU - Kandemir FM AU - Şimşek H AU - Çelik Samancı T AU - Kandemir Ö PY - 2026 JO - Molecular and cellular biochemistry DO - 10.1007/s11010-026-05748-y UR - https://doi.org/10.1007/s11010-026-05748-y ER -
APA
FM, K., H, Ş., T, Ç. S., & Ö, K. (2026). 18β-glycyrrhetinic acid ameliorates ifosfamide-induced nephrotoxicity by suppressing Wnt3a/IL-17A-driven inflammatory signaling, ER stress, and apoptosis while preserving ZO-1 integrity.. Molecular and cellular biochemistry. https://doi.org/10.1007/s11010-026-05748-y
Source records
- pubmed · retrieved 2026-09-26T11:11:04.114Z