Endothelial S1PR3 downregulation exacerbates pressure overload-induced cardiac remodeling by promoting fibrosis and inflammation.
- DOI
- 10.1007/s11427-025-3508-5
- Published
- 2026 Sep 8
- Container
- Science China. Life sciences
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1007/s11427-025-3508-5,
title = {Endothelial S1PR3 downregulation exacerbates pressure overload-induced cardiac remodeling by promoting fibrosis and inflammation.},
author = {Yang X and Cheng K and Wang L and Chen C and Wang DW and Jiang J},
year = {2026},
journal = {Science China. Life sciences},
doi = {10.1007/s11427-025-3508-5},
url = {https://doi.org/10.1007/s11427-025-3508-5}
}RIS
TY - JOUR TI - Endothelial S1PR3 downregulation exacerbates pressure overload-induced cardiac remodeling by promoting fibrosis and inflammation. AU - Yang X AU - Cheng K AU - Wang L AU - Chen C AU - Wang DW AU - Jiang J PY - 2026 JO - Science China. Life sciences DO - 10.1007/s11427-025-3508-5 UR - https://doi.org/10.1007/s11427-025-3508-5 ER -
APA
X, Y., K, C., L, W., C, C., DW, W., & J, J. (2026). Endothelial S1PR3 downregulation exacerbates pressure overload-induced cardiac remodeling by promoting fibrosis and inflammation.. Science China. Life sciences. https://doi.org/10.1007/s11427-025-3508-5
Source records
- pubmed · retrieved 2026-09-26T21:48:47.249Z