Lipoprotein apheresis is an optimal therapeutic option to reduce increased Lp(a) levels.

Schettler VJJ, Neumann CL, Peter C, Zimmermann T, Julius U, Hohenstein B, Roeseler E, Heigl F, Grützmacher P, Blume H, Klingel R, Vogt A, Scientific Board of GLAR for the German Apheresis Working Group

Open source

DOI
10.1007/s11789-019-00094-4
Published
2019 Apr
Container
Clinical research in cardiology supplements
Publisher
Not recorded
Open access
no

Credibility signals

limited evidence Score 43/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1007/s11789-019-00094-4,
  title = {Lipoprotein apheresis is an optimal therapeutic option to reduce increased Lp(a) levels.},
  author = {Schettler VJJ and Neumann CL and Peter C and Zimmermann T and Julius U and Hohenstein B and Roeseler E and Heigl F and Grützmacher P and Blume H and Klingel R and Vogt A and Scientific Board of GLAR for the German Apheresis Working Group},
  year = {2019},
  journal = {Clinical research in cardiology supplements},
  doi = {10.1007/s11789-019-00094-4},
  url = {https://doi.org/10.1007/s11789-019-00094-4}
}

RIS

TY  - JOUR
TI  - Lipoprotein apheresis is an optimal therapeutic option to reduce increased Lp(a) levels.
AU  - Schettler VJJ
AU  - Neumann CL
AU  - Peter C
AU  - Zimmermann T
AU  - Julius U
AU  - Hohenstein B
AU  - Roeseler E
AU  - Heigl F
AU  - Grützmacher P
AU  - Blume H
AU  - Klingel R
AU  - Vogt A
AU  - Scientific Board of GLAR for the German Apheresis Working Group
PY  - 2019
JO  - Clinical research in cardiology supplements
DO  - 10.1007/s11789-019-00094-4
UR  - https://doi.org/10.1007/s11789-019-00094-4
ER  - 

APA

VJJ, S., CL, N., C, P., T, Z., U, J., B, H., E, R., F, H., P, G., H, B., R, K., A, V., & Group, S. B. O. G. F. T. G. A. W. (2019). Lipoprotein apheresis is an optimal therapeutic option to reduce increased Lp(a) levels.. Clinical research in cardiology supplements. https://doi.org/10.1007/s11789-019-00094-4

Source records