Point mutations abolish 11 beta-hydroxysteroid dehydrogenase type II activity in three families with the congenital syndrome of apparent mineralocorticoid excess.
- DOI
- 10.1016/0303-7207(96)03787-2
- Published
- 1996 May 17
- Container
- Molecular and cellular endocrinology
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/0303-7207-96-03787-2,
title = {Point mutations abolish 11 beta-hydroxysteroid dehydrogenase type II activity in three families with the congenital syndrome of apparent mineralocorticoid excess.},
author = {Ferrari P and Obeyesekere VR and Li K and Wilson RC and New MI and Funder JW and Krozowski ZS},
year = {1996},
journal = {Molecular and cellular endocrinology},
doi = {10.1016/0303-7207(96)03787-2},
url = {https://doi.org/10.1016/0303-7207(96)03787-2}
}RIS
TY - JOUR TI - Point mutations abolish 11 beta-hydroxysteroid dehydrogenase type II activity in three families with the congenital syndrome of apparent mineralocorticoid excess. AU - Ferrari P AU - Obeyesekere VR AU - Li K AU - Wilson RC AU - New MI AU - Funder JW AU - Krozowski ZS PY - 1996 JO - Molecular and cellular endocrinology DO - 10.1016/0303-7207(96)03787-2 UR - https://doi.org/10.1016/0303-7207(96)03787-2 ER -
APA
P, F., VR, O., K, L., RC, W., MI, N., JW, F., & ZS, K. (1996). Point mutations abolish 11 beta-hydroxysteroid dehydrogenase type II activity in three families with the congenital syndrome of apparent mineralocorticoid excess.. Molecular and cellular endocrinology. https://doi.org/10.1016/0303-7207(96)03787-2
Source records
- pubmed · retrieved 2026-09-26T01:44:47.157Z