BIRC3 and NOC2L synergistically promote P53 acetylation to accelerate necroptosis in sepsis-associated acute kidney injury.
- DOI
- 10.1016/j.bbadis.2026.168202
- Published
- 2026 Jun
- Container
- Biochimica et biophysica acta. Molecular basis of disease
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.bbadis.2026.168202,
title = {BIRC3 and NOC2L synergistically promote P53 acetylation to accelerate necroptosis in sepsis-associated acute kidney injury.},
author = {Ma X and Wang Y and Diao R and Yin M and Shen Q and Lv W and Xu M and Jin Y},
year = {2026},
journal = {Biochimica et biophysica acta. Molecular basis of disease},
doi = {10.1016/j.bbadis.2026.168202},
url = {https://doi.org/10.1016/j.bbadis.2026.168202}
}RIS
TY - JOUR TI - BIRC3 and NOC2L synergistically promote P53 acetylation to accelerate necroptosis in sepsis-associated acute kidney injury. AU - Ma X AU - Wang Y AU - Diao R AU - Yin M AU - Shen Q AU - Lv W AU - Xu M AU - Jin Y PY - 2026 JO - Biochimica et biophysica acta. Molecular basis of disease DO - 10.1016/j.bbadis.2026.168202 UR - https://doi.org/10.1016/j.bbadis.2026.168202 ER -
APA
X, M., Y, W., R, D., M, Y., Q, S., W, L., M, X., & Y, J. (2026). BIRC3 and NOC2L synergistically promote P53 acetylation to accelerate necroptosis in sepsis-associated acute kidney injury.. Biochimica et biophysica acta. Molecular basis of disease. https://doi.org/10.1016/j.bbadis.2026.168202
Source records
- pubmed · retrieved 2026-09-26T05:43:38.575Z