Intranasal bFGF‑Els alleviate doxorubicin‑induced cognitive deficits in mice by modulating the PI3K/AKT pathway and suppressing oxidative stress, neuroinflammation, and apoptosis
- DOI
- 10.1016/j.bbi.2026.106983
- Published
- 2027-01
- Container
- Brain, Behavior, and Immunity
- Publisher
- Elsevier BV
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.bbi.2026.106983,
title = {Intranasal bFGF‑Els alleviate doxorubicin‑induced cognitive deficits in mice by modulating the PI3K/AKT pathway and suppressing oxidative stress, neuroinflammation, and apoptosis},
author = {Ming Zhang and Wen-yue He and Wei-cheng Ma and Shu-fang Yu},
year = {2027},
journal = {Brain, Behavior, and Immunity},
doi = {10.1016/j.bbi.2026.106983},
url = {https://doi.org/10.1016/j.bbi.2026.106983}
}RIS
TY - JOUR TI - Intranasal bFGF‑Els alleviate doxorubicin‑induced cognitive deficits in mice by modulating the PI3K/AKT pathway and suppressing oxidative stress, neuroinflammation, and apoptosis AU - Ming Zhang AU - Wen-yue He AU - Wei-cheng Ma AU - Shu-fang Yu PY - 2027 JO - Brain, Behavior, and Immunity DO - 10.1016/j.bbi.2026.106983 UR - https://doi.org/10.1016/j.bbi.2026.106983 ER -
APA
Zhang, M., He, W., Ma, W., & Yu, S. (2027). Intranasal bFGF‑Els alleviate doxorubicin‑induced cognitive deficits in mice by modulating the PI3K/AKT pathway and suppressing oxidative stress, neuroinflammation, and apoptosis. Brain, Behavior, and Immunity. https://doi.org/10.1016/j.bbi.2026.106983
Source records
- crossref · retrieved 2026-09-25T02:36:12.320Z