Prolonged depletion of renal tubular thioredoxin following severe acute kidney injury is associated with transition to chronic kidney disease via G2/M cell cycle arrest.

Nishikawa S, Kasuno K, Nishimori K, Shimamoto Y, Nishikawa Y, Kobayashi M, Yokoi S, Takahashi N, Kimura H, Ono T, Muso E, Yoshida H, Irani K, Yodoi J, Namba F, Iwano M, Toyama T

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DOI
10.1016/j.bbrc.2025.151425
Published
2025 Mar 25
Container
Biochemical and biophysical research communications
Publisher
Not recorded
Open access
no

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BibTeX

@article{allodium:10.1016/j.bbrc.2025.151425,
  title = {Prolonged depletion of renal tubular thioredoxin following severe acute kidney injury is associated with transition to chronic kidney disease via G2/M cell cycle arrest.},
  author = {Nishikawa S and Kasuno K and Nishimori K and Shimamoto Y and Nishikawa Y and Kobayashi M and Yokoi S and Takahashi N and Kimura H and Ono T and Muso E and Yoshida H and Irani K and Yodoi J and Namba F and Iwano M and Toyama T},
  year = {2025},
  journal = {Biochemical and biophysical research communications},
  doi = {10.1016/j.bbrc.2025.151425},
  url = {https://doi.org/10.1016/j.bbrc.2025.151425}
}

RIS

TY  - JOUR
TI  - Prolonged depletion of renal tubular thioredoxin following severe acute kidney injury is associated with transition to chronic kidney disease via G2/M cell cycle arrest.
AU  - Nishikawa S
AU  - Kasuno K
AU  - Nishimori K
AU  - Shimamoto Y
AU  - Nishikawa Y
AU  - Kobayashi M
AU  - Yokoi S
AU  - Takahashi N
AU  - Kimura H
AU  - Ono T
AU  - Muso E
AU  - Yoshida H
AU  - Irani K
AU  - Yodoi J
AU  - Namba F
AU  - Iwano M
AU  - Toyama T
PY  - 2025
JO  - Biochemical and biophysical research communications
DO  - 10.1016/j.bbrc.2025.151425
UR  - https://doi.org/10.1016/j.bbrc.2025.151425
ER  - 

APA

S, N., K, K., K, N., Y, S., Y, N., M, K., S, Y., N, T., H, K., T, O., E, M., H, Y., K, I., J, Y., F, N., M, I., & T, T. (2025). Prolonged depletion of renal tubular thioredoxin following severe acute kidney injury is associated with transition to chronic kidney disease via G2/M cell cycle arrest.. Biochemical and biophysical research communications. https://doi.org/10.1016/j.bbrc.2025.151425

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