Hepatocyte SLC31A1-dependent copper accumulation contributes to hepatic glucose and lipid metabolic disturbances through AMPKα1 inactivation.
- DOI
- 10.1016/j.bbrep.2026.102787
- Published
- 2026 Dec
- Container
- Biochemistry and biophysics reports
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1016/j.bbrep.2026.102787,
title = {Hepatocyte SLC31A1-dependent copper accumulation contributes to hepatic glucose and lipid metabolic disturbances through AMPKα1 inactivation.},
author = {Cheng YQ and Wei M and Yang L and Zhuang YY and Lu QB},
year = {2026},
journal = {Biochemistry and biophysics reports},
doi = {10.1016/j.bbrep.2026.102787},
url = {https://doi.org/10.1016/j.bbrep.2026.102787}
}RIS
TY - JOUR TI - Hepatocyte SLC31A1-dependent copper accumulation contributes to hepatic glucose and lipid metabolic disturbances through AMPKα1 inactivation. AU - Cheng YQ AU - Wei M AU - Yang L AU - Zhuang YY AU - Lu QB PY - 2026 JO - Biochemistry and biophysics reports DO - 10.1016/j.bbrep.2026.102787 UR - https://doi.org/10.1016/j.bbrep.2026.102787 ER -
APA
YQ, C., M, W., L, Y., YY, Z., & QB, L. (2026). Hepatocyte SLC31A1-dependent copper accumulation contributes to hepatic glucose and lipid metabolic disturbances through AMPKα1 inactivation.. Biochemistry and biophysics reports. https://doi.org/10.1016/j.bbrep.2026.102787
Source records
- pubmed · retrieved 2026-09-26T06:26:21.922Z
- europe-pmc · retrieved 2026-09-26T06:26:21.933Z
- doaj · retrieved 2026-09-26T06:26:21.920Z