Pharmacological antagonism of HTR2B reduces ERK and STAT3 activation and prevents renal fibrosis during the development of Adenine-Induced CKD in rats.
- DOI
- 10.1016/j.bcp.2026.118482
- Published
- 2026-09-16
- Container
- Biochem Pharmacol
- Publisher
- Not recorded
- Open access
- no
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Cite this work
BibTeX
@article{allodium:10.1016/j.bcp.2026.118482,
title = {Pharmacological antagonism of HTR2B reduces ERK and STAT3 activation and prevents renal fibrosis during the development of Adenine-Induced CKD in rats.},
author = {McAlister KW and Scholpa NE and Janda J and Schnellmann RG.},
year = {2026},
journal = {Biochem Pharmacol},
doi = {10.1016/j.bcp.2026.118482},
url = {https://doi.org/10.1016/j.bcp.2026.118482}
}RIS
TY - JOUR TI - Pharmacological antagonism of HTR2B reduces ERK and STAT3 activation and prevents renal fibrosis during the development of Adenine-Induced CKD in rats. AU - McAlister KW AU - Scholpa NE AU - Janda J AU - Schnellmann RG. PY - 2026 JO - Biochem Pharmacol DO - 10.1016/j.bcp.2026.118482 UR - https://doi.org/10.1016/j.bcp.2026.118482 ER -
APA
KW, M., NE, S., J, J., & RG., S. (2026). Pharmacological antagonism of HTR2B reduces ERK and STAT3 activation and prevents renal fibrosis during the development of Adenine-Induced CKD in rats.. Biochem Pharmacol. https://doi.org/10.1016/j.bcp.2026.118482
Source records
- europe-pmc · retrieved 2026-09-25T01:58:54.792Z