A cell type-specific switch in GluN2D NMDA receptors drives cortical circuit hyperexcitability and sensory-driven cortical synchronization deficits in a Fragile X Syndrome model.
- DOI
- 10.1016/j.biopsych.2026.09.007
- Published
- 2026 Sep 21
- Container
- Biological psychiatry
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.biopsych.2026.09.007,
title = {A cell type-specific switch in GluN2D NMDA receptors drives cortical circuit hyperexcitability and sensory-driven cortical synchronization deficits in a Fragile X Syndrome model.},
author = {Shedd A and Jonak C and Chan L and Assad S and Dravid S and Gibson J and Binder D and Huber K},
year = {2026},
journal = {Biological psychiatry},
doi = {10.1016/j.biopsych.2026.09.007},
url = {https://doi.org/10.1016/j.biopsych.2026.09.007}
}RIS
TY - JOUR TI - A cell type-specific switch in GluN2D NMDA receptors drives cortical circuit hyperexcitability and sensory-driven cortical synchronization deficits in a Fragile X Syndrome model. AU - Shedd A AU - Jonak C AU - Chan L AU - Assad S AU - Dravid S AU - Gibson J AU - Binder D AU - Huber K PY - 2026 JO - Biological psychiatry DO - 10.1016/j.biopsych.2026.09.007 UR - https://doi.org/10.1016/j.biopsych.2026.09.007 ER -
APA
A, S., C, J., L, C., S, A., S, D., J, G., D, B., & K, H. (2026). A cell type-specific switch in GluN2D NMDA receptors drives cortical circuit hyperexcitability and sensory-driven cortical synchronization deficits in a Fragile X Syndrome model.. Biological psychiatry. https://doi.org/10.1016/j.biopsych.2026.09.007
Source records
- pubmed · retrieved 2026-09-25T13:56:46.516Z