Biphasic tRF-31 dynamics regulate HIPK2-mediated dual-track lens epithelial injury under oxidative stress in age-related cataract.
- DOI
- 10.1016/j.exer.2026.111292
- Published
- 2026 Dec
- Container
- Experimental eye research
- Publisher
- Not recorded
- Open access
- no
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Cite this work
BibTeX
@article{allodium:10.1016/j.exer.2026.111292,
title = {Biphasic tRF-31 dynamics regulate HIPK2-mediated dual-track lens epithelial injury under oxidative stress in age-related cataract.},
author = {Pan Z and Jin Y and Zhang C and Yu Y},
year = {2026},
journal = {Experimental eye research},
doi = {10.1016/j.exer.2026.111292},
url = {https://doi.org/10.1016/j.exer.2026.111292}
}RIS
TY - JOUR TI - Biphasic tRF-31 dynamics regulate HIPK2-mediated dual-track lens epithelial injury under oxidative stress in age-related cataract. AU - Pan Z AU - Jin Y AU - Zhang C AU - Yu Y PY - 2026 JO - Experimental eye research DO - 10.1016/j.exer.2026.111292 UR - https://doi.org/10.1016/j.exer.2026.111292 ER -
APA
Z, P., Y, J., C, Z., & Y, Y. (2026). Biphasic tRF-31 dynamics regulate HIPK2-mediated dual-track lens epithelial injury under oxidative stress in age-related cataract.. Experimental eye research. https://doi.org/10.1016/j.exer.2026.111292
Source records
- pubmed · retrieved 2026-11-10T11:35:32.367Z
- europe-pmc · retrieved 2026-11-10T11:35:32.385Z