The iron overload caused by non-enzymatically glycated transferrin triggers oxidative stress and ferroptosis by regulating the PI3/AKT signalling pathway and the expression of Nrf2 to promote renal damage in diabetes.
- DOI
- 10.1016/j.freeradbiomed.2026.08.030
- Published
- 2026 Aug 19
- Container
- Free radical biology & medicine
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.freeradbiomed.2026.08.030,
title = {The iron overload caused by non-enzymatically glycated transferrin triggers oxidative stress and ferroptosis by regulating the PI3/AKT signalling pathway and the expression of Nrf2 to promote renal damage in diabetes.},
author = {Zhao P and Pan B and Lv X and Ma L and Liu J},
year = {2026},
journal = {Free radical biology \& medicine},
doi = {10.1016/j.freeradbiomed.2026.08.030},
url = {https://doi.org/10.1016/j.freeradbiomed.2026.08.030}
}RIS
TY - JOUR TI - The iron overload caused by non-enzymatically glycated transferrin triggers oxidative stress and ferroptosis by regulating the PI3/AKT signalling pathway and the expression of Nrf2 to promote renal damage in diabetes. AU - Zhao P AU - Pan B AU - Lv X AU - Ma L AU - Liu J PY - 2026 JO - Free radical biology & medicine DO - 10.1016/j.freeradbiomed.2026.08.030 UR - https://doi.org/10.1016/j.freeradbiomed.2026.08.030 ER -
APA
P, Z., B, P., X, L., L, M., & J, L. (2026). The iron overload caused by non-enzymatically glycated transferrin triggers oxidative stress and ferroptosis by regulating the PI3/AKT signalling pathway and the expression of Nrf2 to promote renal damage in diabetes.. Free radical biology & medicine. https://doi.org/10.1016/j.freeradbiomed.2026.08.030
Source records
- pubmed · retrieved 2026-09-27T13:22:27.151Z