Dimethyl fumarate ameliorates amyloid-beta generated cognitive deficits by inhibiting RIPK1/RIPK3/MLKL necroptosis signalling in rats.

Guin S, Jangli A, Dandekar MP

DOI: 10.1016/j.intimp.2026.117407
Published: 2026 Sep 15
Container: International immunopharmacology
Publisher: Not recorded
Open access: unknown

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Cite this work

BibTeX

@article{allodium:10.1016/j.intimp.2026.117407,
  title = {Dimethyl fumarate ameliorates amyloid-beta generated cognitive deficits by inhibiting RIPK1/RIPK3/MLKL necroptosis signalling in rats.},
  author = {Guin S and Jangli A and Dandekar MP},
  year = {2026},
  journal = {International immunopharmacology},
  doi = {10.1016/j.intimp.2026.117407},
  url = {https://doi.org/10.1016/j.intimp.2026.117407}
}

RIS

TY  - JOUR
TI  - Dimethyl fumarate ameliorates amyloid-beta generated cognitive deficits by inhibiting RIPK1/RIPK3/MLKL necroptosis signalling in rats.
AU  - Guin S
AU  - Jangli A
AU  - Dandekar MP
PY  - 2026
JO  - International immunopharmacology
DO  - 10.1016/j.intimp.2026.117407
UR  - https://doi.org/10.1016/j.intimp.2026.117407
ER  -

APA

S, G., A, J., & MP, D. (2026). Dimethyl fumarate ameliorates amyloid-beta generated cognitive deficits by inhibiting RIPK1/RIPK3/MLKL necroptosis signalling in rats.. International immunopharmacology. https://doi.org/10.1016/j.intimp.2026.117407

Source records

pubmed · retrieved 2026-09-25T11:27:30.354Z