IGF2BP3 drives macrophage M1 polarization to promote intracranial aneurysm rupture through m6A-mediated NPY1R stabilization.
- DOI
- 10.1016/j.peptides.2026.171522
- Published
- 2026 Sep 22
- Container
- Peptides
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.peptides.2026.171522,
title = {IGF2BP3 drives macrophage M1 polarization to promote intracranial aneurysm rupture through m6A-mediated NPY1R stabilization.},
author = {He J and Tang T and Duan Y and Liang R and Zhou M and Wang B and Jiang Y},
year = {2026},
journal = {Peptides},
doi = {10.1016/j.peptides.2026.171522},
url = {https://doi.org/10.1016/j.peptides.2026.171522}
}RIS
TY - JOUR TI - IGF2BP3 drives macrophage M1 polarization to promote intracranial aneurysm rupture through m6A-mediated NPY1R stabilization. AU - He J AU - Tang T AU - Duan Y AU - Liang R AU - Zhou M AU - Wang B AU - Jiang Y PY - 2026 JO - Peptides DO - 10.1016/j.peptides.2026.171522 UR - https://doi.org/10.1016/j.peptides.2026.171522 ER -
APA
J, H., T, T., Y, D., R, L., M, Z., B, W., & Y, J. (2026). IGF2BP3 drives macrophage M1 polarization to promote intracranial aneurysm rupture through m6A-mediated NPY1R stabilization.. Peptides. https://doi.org/10.1016/j.peptides.2026.171522
Source records
- pubmed · retrieved 2026-09-25T16:25:02.655Z