Defective lysosomal acidification promotes chondrocyte senescence and autophagic flux stagnation in geriatric osteoarthritis.
- DOI
- 10.1016/j.tice.2026.103767
- Published
- 2026 Jul 9
- Container
- Tissue & cell
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.tice.2026.103767,
title = {Defective lysosomal acidification promotes chondrocyte senescence and autophagic flux stagnation in geriatric osteoarthritis.},
author = {Lakshmanan DK and Thilagar S and Shanmugam A and Kalidass B and Ravichandran G},
year = {2026},
journal = {Tissue \& cell},
doi = {10.1016/j.tice.2026.103767},
url = {https://doi.org/10.1016/j.tice.2026.103767}
}RIS
TY - JOUR TI - Defective lysosomal acidification promotes chondrocyte senescence and autophagic flux stagnation in geriatric osteoarthritis. AU - Lakshmanan DK AU - Thilagar S AU - Shanmugam A AU - Kalidass B AU - Ravichandran G PY - 2026 JO - Tissue & cell DO - 10.1016/j.tice.2026.103767 UR - https://doi.org/10.1016/j.tice.2026.103767 ER -
APA
DK, L., S, T., A, S., B, K., & G, R. (2026). Defective lysosomal acidification promotes chondrocyte senescence and autophagic flux stagnation in geriatric osteoarthritis.. Tissue & cell. https://doi.org/10.1016/j.tice.2026.103767
Source records
- pubmed · retrieved 2026-09-27T14:34:15.906Z