Effects of Ivabradine (I(f) channel inhibitor) and N-Acetylcysteine against isoprenaline-induced myocardial injury: Role of Nrf2/HO-1 signaling and inflammation suppression.
- DOI
- 10.1016/j.tice.2026.103806
- Published
- 2026 Jul 23
- Container
- Tissue & cell
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.tice.2026.103806,
title = {Effects of Ivabradine (I(f) channel inhibitor) and N-Acetylcysteine against isoprenaline-induced myocardial injury: Role of Nrf2/HO-1 signaling and inflammation suppression.},
author = {Ali LS and Aladeeb NM and Hamed WHE and El-Hanafy AA and Eldesoqui M and Al-Wakeel G},
year = {2026},
journal = {Tissue \& cell},
doi = {10.1016/j.tice.2026.103806},
url = {https://doi.org/10.1016/j.tice.2026.103806}
}RIS
TY - JOUR TI - Effects of Ivabradine (I(f) channel inhibitor) and N-Acetylcysteine against isoprenaline-induced myocardial injury: Role of Nrf2/HO-1 signaling and inflammation suppression. AU - Ali LS AU - Aladeeb NM AU - Hamed WHE AU - El-Hanafy AA AU - Eldesoqui M AU - Al-Wakeel G PY - 2026 JO - Tissue & cell DO - 10.1016/j.tice.2026.103806 UR - https://doi.org/10.1016/j.tice.2026.103806 ER -
APA
LS, A., NM, A., WHE, H., AA, E., M, E., & G, A. (2026). Effects of Ivabradine (I(f) channel inhibitor) and N-Acetylcysteine against isoprenaline-induced myocardial injury: Role of Nrf2/HO-1 signaling and inflammation suppression.. Tissue & cell. https://doi.org/10.1016/j.tice.2026.103806
Source records
- pubmed · retrieved 2026-09-26T17:58:16.800Z