Targeting RNase H2: A dual-mechanism strategy to elevate replication stress, DNA damage, and antitumor immunity in TNBC
- DOI
- 10.1016/j.xcrm.2026.102750
- Published
- 2026-05
- Container
- Cell Reports Medicine
- Publisher
- Elsevier BV
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.xcrm.2026.102750,
title = {Targeting RNase H2: A dual-mechanism strategy to elevate replication stress, DNA damage, and antitumor immunity in TNBC},
author = {Thai Quynh Anh Nguyen and Jing Zhang and Hui Dai and Aysegul Murat and Yong Du and Daniel J. McGrail and Funda Meric-Bernstam and Shiaw-Yih Lin},
year = {2026},
journal = {Cell Reports Medicine},
doi = {10.1016/j.xcrm.2026.102750},
url = {https://doi.org/10.1016/j.xcrm.2026.102750}
}RIS
TY - JOUR TI - Targeting RNase H2: A dual-mechanism strategy to elevate replication stress, DNA damage, and antitumor immunity in TNBC AU - Thai Quynh Anh Nguyen AU - Jing Zhang AU - Hui Dai AU - Aysegul Murat AU - Yong Du AU - Daniel J. McGrail AU - Funda Meric-Bernstam AU - Shiaw-Yih Lin PY - 2026 JO - Cell Reports Medicine DO - 10.1016/j.xcrm.2026.102750 UR - https://doi.org/10.1016/j.xcrm.2026.102750 ER -
APA
Nguyen, T. Q. A., Zhang, J., Dai, H., Murat, A., Du, Y., McGrail, D. J., Meric-Bernstam, F., & Lin, S. (2026). Targeting RNase H2: A dual-mechanism strategy to elevate replication stress, DNA damage, and antitumor immunity in TNBC. Cell Reports Medicine. https://doi.org/10.1016/j.xcrm.2026.102750
Source records
- crossref · retrieved 2026-09-26T06:36:52.279Z