Fatty-acid-based antimiR-23b delivery in the DMSXL model: A potential therapeutic strategy for brain dysfunction in myotonic dystrophy type 1
- DOI
- 10.1016/j.xcrm.2026.102929
- Published
- 2026-08
- Container
- Cell Reports Medicine
- Publisher
- Elsevier BV
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1016/j.xcrm.2026.102929,
title = {Fatty-acid-based antimiR-23b delivery in the DMSXL model: A potential therapeutic strategy for brain dysfunction in myotonic dystrophy type 1},
author = {Diego Piqueras-Losilla and Andrea Garcia-Rey and Aline Huguet-Lachon and Argimiro Mayoral-Olmos and Isabel Campillo and Melanie Nufer and Mouli Chakraborty and Ana Díaz-Maqueda and Nuria Barquero and Anchel Gonzalez-Barriga and José Martinez-Hernandez and María Gracia de Garnica García and Geneviève Gourdon and Ruben Artero and Beatriz Llamusí and Estefanía Cerro-Herreros},
year = {2026},
journal = {Cell Reports Medicine},
doi = {10.1016/j.xcrm.2026.102929},
url = {https://doi.org/10.1016/j.xcrm.2026.102929}
}RIS
TY - JOUR TI - Fatty-acid-based antimiR-23b delivery in the DMSXL model: A potential therapeutic strategy for brain dysfunction in myotonic dystrophy type 1 AU - Diego Piqueras-Losilla AU - Andrea Garcia-Rey AU - Aline Huguet-Lachon AU - Argimiro Mayoral-Olmos AU - Isabel Campillo AU - Melanie Nufer AU - Mouli Chakraborty AU - Ana Díaz-Maqueda AU - Nuria Barquero AU - Anchel Gonzalez-Barriga AU - José Martinez-Hernandez AU - María Gracia de Garnica García AU - Geneviève Gourdon AU - Ruben Artero AU - Beatriz Llamusí AU - Estefanía Cerro-Herreros PY - 2026 JO - Cell Reports Medicine DO - 10.1016/j.xcrm.2026.102929 UR - https://doi.org/10.1016/j.xcrm.2026.102929 ER -
APA
Piqueras-Losilla, D., Garcia-Rey, A., Huguet-Lachon, A., Mayoral-Olmos, A., Campillo, I., Nufer, M., Chakraborty, M., Díaz-Maqueda, A., Barquero, N., Gonzalez-Barriga, A., Martinez-Hernandez, J., García, M. G. D. G., Gourdon, G., Artero, R., Llamusí, B., & Cerro-Herreros, E. (2026). Fatty-acid-based antimiR-23b delivery in the DMSXL model: A potential therapeutic strategy for brain dysfunction in myotonic dystrophy type 1. Cell Reports Medicine. https://doi.org/10.1016/j.xcrm.2026.102929
Source records
- crossref · retrieved 2026-09-25T14:25:39.120Z