The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells

Hanna Erdmann, Jochen Behrends, Kristina Ritter, Alexandra Hölscher, Johanna Volz, Ida Rosenkrands, Christoph Hölscher

Open source

DOI
10.1038/s41385-018-0026-3
Published
2018-07
Container
Mucosal Immunology
Publisher
Elsevier BV
Open access
unknown

Credibility signals

uncertain Score 64/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1038/s41385-018-0026-3,
  title = {The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells},
  author = {Hanna Erdmann and Jochen Behrends and Kristina Ritter and Alexandra Hölscher and Johanna Volz and Ida Rosenkrands and Christoph Hölscher},
  year = {2018},
  journal = {Mucosal Immunology},
  doi = {10.1038/s41385-018-0026-3},
  url = {https://doi.org/10.1038/s41385-018-0026-3}
}

RIS

TY  - JOUR
TI  - The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells
AU  - Hanna Erdmann
AU  - Jochen Behrends
AU  - Kristina Ritter
AU  - Alexandra Hölscher
AU  - Johanna Volz
AU  - Ida Rosenkrands
AU  - Christoph Hölscher
PY  - 2018
JO  - Mucosal Immunology
DO  - 10.1038/s41385-018-0026-3
UR  - https://doi.org/10.1038/s41385-018-0026-3
ER  - 

APA

Erdmann, H., Behrends, J., Ritter, K., Hölscher, A., Volz, J., Rosenkrands, I., & Hölscher, C. (2018). The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells. Mucosal Immunology. https://doi.org/10.1038/s41385-018-0026-3

Source records