A RAS-CaMKKβ-AMPKα2 pathway promotes senescence by licensing post-translational activation of C/EBPβ through a novel 3'UTR mechanism.

Basu SK, Gonit M, Salotti J, Chen J, Bhat A, Gorospe M, Viollet B, Claffey KP, Johnson PF

DOI: 10.1038/s41388-018-0190-7
Published: 2018 Jun
Container: Oncogene
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.1038/s41388-018-0190-7,
  title = {A RAS-CaMKKβ-AMPKα2 pathway promotes senescence by licensing post-translational activation of C/EBPβ through a novel 3'UTR mechanism.},
  author = {Basu SK and Gonit M and Salotti J and Chen J and Bhat A and Gorospe M and Viollet B and Claffey KP and Johnson PF},
  year = {2018},
  journal = {Oncogene},
  doi = {10.1038/s41388-018-0190-7},
  url = {https://doi.org/10.1038/s41388-018-0190-7}
}

RIS

TY  - JOUR
TI  - A RAS-CaMKKβ-AMPKα2 pathway promotes senescence by licensing post-translational activation of C/EBPβ through a novel 3'UTR mechanism.
AU  - Basu SK
AU  - Gonit M
AU  - Salotti J
AU  - Chen J
AU  - Bhat A
AU  - Gorospe M
AU  - Viollet B
AU  - Claffey KP
AU  - Johnson PF
PY  - 2018
JO  - Oncogene
DO  - 10.1038/s41388-018-0190-7
UR  - https://doi.org/10.1038/s41388-018-0190-7
ER  -

APA

SK, B., M, G., J, S., J, C., A, B., M, G., B, V., KP, C., & PF, J. (2018). A RAS-CaMKKβ-AMPKα2 pathway promotes senescence by licensing post-translational activation of C/EBPβ through a novel 3'UTR mechanism.. Oncogene. https://doi.org/10.1038/s41388-018-0190-7

Source records

pubmed · retrieved 2026-09-26T00:15:55.012Z