HMGB1 directly activates BAP1 transcription to sustain tumor growth in mesothelioma
- DOI
- 10.1038/s41418-026-01892-6
- Published
- 2026-10-23
- Container
- Cell Death & Differentiation
- Publisher
- Springer Science and Business Media LLC
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1038/s41418-026-01892-6,
title = {HMGB1 directly activates BAP1 transcription to sustain tumor growth in mesothelioma},
author = {Flavia Novelli and Lydia Giannakou and Zhanwei Wang and Jin-Hee Kim and Qian Wang and Miao Feng and Zeying Li and Cristina Favaron and Filippo Maldini and Joelle S. Suarez and Mika Tanji and Alicia A. Zolondick and Angela Bononi and Linda MacPherson and Euisung Hong and Xin Chen and Youping Deng and Tak W. Mak and Harvey I. Pass and Jose N. Onuchic and Michele Carbone and Haining Yang},
year = {2026},
journal = {Cell Death \& Differentiation},
doi = {10.1038/s41418-026-01892-6},
url = {https://doi.org/10.1038/s41418-026-01892-6}
}RIS
TY - JOUR TI - HMGB1 directly activates BAP1 transcription to sustain tumor growth in mesothelioma AU - Flavia Novelli AU - Lydia Giannakou AU - Zhanwei Wang AU - Jin-Hee Kim AU - Qian Wang AU - Miao Feng AU - Zeying Li AU - Cristina Favaron AU - Filippo Maldini AU - Joelle S. Suarez AU - Mika Tanji AU - Alicia A. Zolondick AU - Angela Bononi AU - Linda MacPherson AU - Euisung Hong AU - Xin Chen AU - Youping Deng AU - Tak W. Mak AU - Harvey I. Pass AU - Jose N. Onuchic AU - Michele Carbone AU - Haining Yang PY - 2026 JO - Cell Death & Differentiation DO - 10.1038/s41418-026-01892-6 UR - https://doi.org/10.1038/s41418-026-01892-6 ER -
APA
Novelli, F., Giannakou, L., Wang, Z., Kim, J., Wang, Q., Feng, M., Li, Z., Favaron, C., Maldini, F., Suarez, J. S., Tanji, M., Zolondick, A. A., Bononi, A., MacPherson, L., Hong, E., Chen, X., Deng, Y., Mak, T. W., Pass, H. I., Onuchic, J. N., Carbone, M., & Yang, H. (2026). HMGB1 directly activates BAP1 transcription to sustain tumor growth in mesothelioma. Cell Death & Differentiation. https://doi.org/10.1038/s41418-026-01892-6
Source records
- crossref · retrieved 2026-11-10T13:51:55.346Z