Myelodysplastic Syndrome associated TET2 mutations affect NK cell function and genome methylation
- DOI
- 10.1038/s41467-023-36193-w
- Published
- 2023-02-03
- Container
- Nature Communications
- Publisher
- Springer Science and Business Media LLC
- Open access
- unknown
Credibility signals
uncertain Score 64/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.
Show all credibility signals
- supportingDOI registered: A matching record was returned by Crossref.
- supportingDOI resolves: A matching record was returned by Crossref.
- not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility.
- not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made.
- not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made.
- not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made.
- not scoredROR affiliation: Not checked or no result supplied; no credibility inference made.
- not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete.
- not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete.
- not scoredRetraction Watch correction: No correction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete.
- not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete.
- not scoredOpen access status: Not checked or no result supplied; no credibility inference made.
- not scoredPublication license: Not checked or no result supplied; no credibility inference made.
- not scoredPublication version: A publication version was supplied but is not scored.
- supportingMetadata completeness: All 6 scored descriptive metadata groups are present.
Cite this work
BibTeX
@article{allodium:10.1038/s41467-023-36193-w,
title = {Myelodysplastic Syndrome associated TET2 mutations affect NK cell function and genome methylation},
author = {Maxime Boy and Valeria Bisio and Lin-Pierre Zhao and Fabien Guidez and Bérénice Schell and Emilie Lereclus and Guylaine Henry and Juliette Villemonteix and Fernando Rodrigues-Lima and Katia Gagne and Christelle Retiere and Lise Larcher and Rathana Kim and Emmanuelle Clappier and Marie Sebert and Arsène Mekinian and Olivier Fain and Anne Caignard and Marion Espeli and Karl Balabanian and Antoine Toubert and Pierre Fenaux and Lionel Ades and Nicolas Dulphy},
year = {2023},
journal = {Nature Communications},
doi = {10.1038/s41467-023-36193-w},
url = {https://doi.org/10.1038/s41467-023-36193-w}
}RIS
TY - JOUR TI - Myelodysplastic Syndrome associated TET2 mutations affect NK cell function and genome methylation AU - Maxime Boy AU - Valeria Bisio AU - Lin-Pierre Zhao AU - Fabien Guidez AU - Bérénice Schell AU - Emilie Lereclus AU - Guylaine Henry AU - Juliette Villemonteix AU - Fernando Rodrigues-Lima AU - Katia Gagne AU - Christelle Retiere AU - Lise Larcher AU - Rathana Kim AU - Emmanuelle Clappier AU - Marie Sebert AU - Arsène Mekinian AU - Olivier Fain AU - Anne Caignard AU - Marion Espeli AU - Karl Balabanian AU - Antoine Toubert AU - Pierre Fenaux AU - Lionel Ades AU - Nicolas Dulphy PY - 2023 JO - Nature Communications DO - 10.1038/s41467-023-36193-w UR - https://doi.org/10.1038/s41467-023-36193-w ER -
APA
Boy, M., Bisio, V., Zhao, L., Guidez, F., Schell, B., Lereclus, E., Henry, G., Villemonteix, J., Rodrigues-Lima, F., Gagne, K., Retiere, C., Larcher, L., Kim, R., Clappier, E., Sebert, M., Mekinian, A., Fain, O., Caignard, A., Espeli, M., Balabanian, K., Toubert, A., Fenaux, P., Ades, L., & Dulphy, N. (2023). Myelodysplastic Syndrome associated TET2 mutations affect NK cell function and genome methylation. Nature Communications. https://doi.org/10.1038/s41467-023-36193-w
Source records
- crossref · retrieved 2026-09-26T15:25:03.712Z