β-cell FMO3-produced TMAO prevents NF-κB-mediated senescence and inflammation in ageing and diabetic conditions.

Wang B, Yang Y, Ho YM, Lin H, Yuan J, Long K, Cheng KKY, Li X

Open source

DOI
10.1038/s41467-026-76075-5
Published
2026 Jul 27
Container
Nature communications
Publisher
Not recorded
Open access
yes

Credibility signals

limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1038/s41467-026-76075-5,
  title = {β-cell FMO3-produced TMAO prevents NF-κB-mediated senescence and inflammation in ageing and diabetic conditions.},
  author = {Wang B and Yang Y and Ho YM and Lin H and Yuan J and Long K and Cheng KKY and Li X},
  year = {2026},
  journal = {Nature communications},
  doi = {10.1038/s41467-026-76075-5},
  url = {https://doi.org/10.1038/s41467-026-76075-5}
}

RIS

TY  - JOUR
TI  - β-cell FMO3-produced TMAO prevents NF-κB-mediated senescence and inflammation in ageing and diabetic conditions.
AU  - Wang B
AU  - Yang Y
AU  - Ho YM
AU  - Lin H
AU  - Yuan J
AU  - Long K
AU  - Cheng KKY
AU  - Li X
PY  - 2026
JO  - Nature communications
DO  - 10.1038/s41467-026-76075-5
UR  - https://doi.org/10.1038/s41467-026-76075-5
ER  - 

APA

B, W., Y, Y., YM, H., H, L., J, Y., K, L., KKY, C., & X, L. (2026). β-cell FMO3-produced TMAO prevents NF-κB-mediated senescence and inflammation in ageing and diabetic conditions.. Nature communications. https://doi.org/10.1038/s41467-026-76075-5

Source records