Enhanced axonal mitochondrial motility and neural activity-induced energy deficits destabilize synaptic transmission in models of bipolar disorder.

Li S, Xiong GJ, Li Z, Sheng ZH

DOI: 10.1038/s41467-026-76722-x
Published: 2026 Aug 22
Container: Nature communications
Publisher: Not recorded
Open access: yes

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BibTeX

@article{allodium:10.1038/s41467-026-76722-x,
  title = {Enhanced axonal mitochondrial motility and neural activity-induced energy deficits destabilize synaptic transmission in models of bipolar disorder.},
  author = {Li S and Xiong GJ and Li Z and Sheng ZH},
  year = {2026},
  journal = {Nature communications},
  doi = {10.1038/s41467-026-76722-x},
  url = {https://doi.org/10.1038/s41467-026-76722-x}
}

RIS

TY  - JOUR
TI  - Enhanced axonal mitochondrial motility and neural activity-induced energy deficits destabilize synaptic transmission in models of bipolar disorder.
AU  - Li S
AU  - Xiong GJ
AU  - Li Z
AU  - Sheng ZH
PY  - 2026
JO  - Nature communications
DO  - 10.1038/s41467-026-76722-x
UR  - https://doi.org/10.1038/s41467-026-76722-x
ER  -

APA

S, L., GJ, X., Z, L., & ZH, S. (2026). Enhanced axonal mitochondrial motility and neural activity-induced energy deficits destabilize synaptic transmission in models of bipolar disorder.. Nature communications. https://doi.org/10.1038/s41467-026-76722-x

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pubmed · retrieved 2026-09-26T01:34:46.380Z