HERC2-UBE3A structure uncovers a domain-motif protein network potentially altered in Angelman and related syndromes.

Demenge A, Lim J, Roc A, Zambo B, Gogl G, Cousido-Siah A, McEwen AG, Bonhoure A, Kostmann C, Fagotto-Kaufmann C, Eberling P, Osz-Papai J, Podjarny A, Howard E, Golzio C, Debant A, Schmidt S, Travé G

Open source

DOI
10.1038/s41467-026-78412-0
Published
2026 Oct 31
Container
Nature communications
Publisher
Not recorded
Open access
unknown

Credibility signals

limited evidence Score 43/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1038/s41467-026-78412-0,
  title = {HERC2-UBE3A structure uncovers a domain-motif protein network potentially altered in Angelman and related syndromes.},
  author = {Demenge A and Lim J and Roc A and Zambo B and Gogl G and Cousido-Siah A and McEwen AG and Bonhoure A and Kostmann C and Fagotto-Kaufmann C and Eberling P and Osz-Papai J and Podjarny A and Howard E and Golzio C and Debant A and Schmidt S and Travé G},
  year = {2026},
  journal = {Nature communications},
  doi = {10.1038/s41467-026-78412-0},
  url = {https://doi.org/10.1038/s41467-026-78412-0}
}

RIS

TY  - JOUR
TI  - HERC2-UBE3A structure uncovers a domain-motif protein network potentially altered in Angelman and related syndromes.
AU  - Demenge A
AU  - Lim J
AU  - Roc A
AU  - Zambo B
AU  - Gogl G
AU  - Cousido-Siah A
AU  - McEwen AG
AU  - Bonhoure A
AU  - Kostmann C
AU  - Fagotto-Kaufmann C
AU  - Eberling P
AU  - Osz-Papai J
AU  - Podjarny A
AU  - Howard E
AU  - Golzio C
AU  - Debant A
AU  - Schmidt S
AU  - Travé G
PY  - 2026
JO  - Nature communications
DO  - 10.1038/s41467-026-78412-0
UR  - https://doi.org/10.1038/s41467-026-78412-0
ER  - 

APA

A, D., J, L., A, R., B, Z., G, G., A, C., AG, M., A, B., C, K., C, F., P, E., J, O., A, P., E, H., C, G., A, D., S, S., & G, T. (2026). HERC2-UBE3A structure uncovers a domain-motif protein network potentially altered in Angelman and related syndromes.. Nature communications. https://doi.org/10.1038/s41467-026-78412-0

Source records