Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical β-catenin-dependent mechanism.
- DOI
- 10.1038/s41598-020-67401-y
- Published
- 2020 Jun 29
- Container
- Scientific reports
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1038/s41598-020-67401-y,
title = {Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical β-catenin-dependent mechanism.},
author = {Ali N and Nguyen CB and Chandrakesan P and Wolf RF and Qu D and May R and Goretsky T and Fazili J and Barrett TA and Li M and Huycke MM and Bronze MS and Houchen CW},
year = {2020},
journal = {Scientific reports},
doi = {10.1038/s41598-020-67401-y},
url = {https://doi.org/10.1038/s41598-020-67401-y}
}RIS
TY - JOUR TI - Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical β-catenin-dependent mechanism. AU - Ali N AU - Nguyen CB AU - Chandrakesan P AU - Wolf RF AU - Qu D AU - May R AU - Goretsky T AU - Fazili J AU - Barrett TA AU - Li M AU - Huycke MM AU - Bronze MS AU - Houchen CW PY - 2020 JO - Scientific reports DO - 10.1038/s41598-020-67401-y UR - https://doi.org/10.1038/s41598-020-67401-y ER -
APA
N, A., CB, N., P, C., RF, W., D, Q., R, M., T, G., J, F., TA, B., M, L., MM, H., MS, B., & CW, H. (2020). Doublecortin-like kinase 1 promotes hepatocyte clonogenicity and oncogenic programming via non-canonical β-catenin-dependent mechanism.. Scientific reports. https://doi.org/10.1038/s41598-020-67401-y
Source records
- pubmed · retrieved 2026-09-26T17:37:25.696Z
- europe-pmc · retrieved 2026-09-26T17:37:25.709Z