Oncolysis of pancreatic tumour cells by a gamma34.5-deleted HSV-1 does not rely upon Ras-activation, but on the PI 3-kinase pathway.

Sarinella F, Calistri A, Sette P, Palù G, Parolin C

DOI: 10.1038/sj.gt.3302770
Published: 2006 Jul
Container: Gene therapy
Publisher: Not recorded
Open access: unknown

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Cite this work

BibTeX

@article{allodium:10.1038/sj.gt.3302770,
  title = {Oncolysis of pancreatic tumour cells by a gamma34.5-deleted HSV-1 does not rely upon Ras-activation, but on the PI 3-kinase pathway.},
  author = {Sarinella F and Calistri A and Sette P and Palù G and Parolin C},
  year = {2006},
  journal = {Gene therapy},
  doi = {10.1038/sj.gt.3302770},
  url = {https://doi.org/10.1038/sj.gt.3302770}
}

RIS

TY  - JOUR
TI  - Oncolysis of pancreatic tumour cells by a gamma34.5-deleted HSV-1 does not rely upon Ras-activation, but on the PI 3-kinase pathway.
AU  - Sarinella F
AU  - Calistri A
AU  - Sette P
AU  - Palù G
AU  - Parolin C
PY  - 2006
JO  - Gene therapy
DO  - 10.1038/sj.gt.3302770
UR  - https://doi.org/10.1038/sj.gt.3302770
ER  -

APA

F, S., A, C., P, S., G, P., & C, P. (2006). Oncolysis of pancreatic tumour cells by a gamma34.5-deleted HSV-1 does not rely upon Ras-activation, but on the PI 3-kinase pathway.. Gene therapy. https://doi.org/10.1038/sj.gt.3302770

Source records

pubmed · retrieved 2026-09-25T11:29:27.361Z