TrxR2 overexpression alleviates inflammation-mediated neuronal death via reducing the oxidative stress and activating the Akt-Parkin pathway.

Gao J, Li Y, Li W, Wang H

DOI: 10.1039/c9tx00076c
Published: 2019 Sep 1
Container: Toxicology research
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.1039/c9tx00076c,
  title = {TrxR2 overexpression alleviates inflammation-mediated neuronal death via reducing the oxidative stress and activating the Akt-Parkin pathway.},
  author = {Gao J and Li Y and Li W and Wang H},
  year = {2019},
  journal = {Toxicology research},
  doi = {10.1039/c9tx00076c},
  url = {https://doi.org/10.1039/c9tx00076c}
}

RIS

TY  - JOUR
TI  - TrxR2 overexpression alleviates inflammation-mediated neuronal death via reducing the oxidative stress and activating the Akt-Parkin pathway.
AU  - Gao J
AU  - Li Y
AU  - Li W
AU  - Wang H
PY  - 2019
JO  - Toxicology research
DO  - 10.1039/c9tx00076c
UR  - https://doi.org/10.1039/c9tx00076c
ER  -

APA

J, G., Y, L., W, L., & H, W. (2019). TrxR2 overexpression alleviates inflammation-mediated neuronal death via reducing the oxidative stress and activating the Akt-Parkin pathway.. Toxicology research. https://doi.org/10.1039/c9tx00076c

Source records

pubmed · retrieved 2026-09-24T20:06:23.462Z