IGFBP2 attenuates high glucose-induced endothelial dysfunction through PI3K/Akt/eNOS pathway activation.

Song X, Zhong W, Chen C, Zheng H, Li Z

DOI: 10.1042/bsr20260182
Published: 2026 Nov 18
Container: Bioscience reports
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.1042/bsr20260182,
  title = {IGFBP2 attenuates high glucose-induced endothelial dysfunction through PI3K/Akt/eNOS pathway activation.},
  author = {Song X and Zhong W and Chen C and Zheng H and Li Z},
  year = {2026},
  journal = {Bioscience reports},
  doi = {10.1042/bsr20260182},
  url = {https://doi.org/10.1042/bsr20260182}
}

RIS

TY  - JOUR
TI  - IGFBP2 attenuates high glucose-induced endothelial dysfunction through PI3K/Akt/eNOS pathway activation.
AU  - Song X
AU  - Zhong W
AU  - Chen C
AU  - Zheng H
AU  - Li Z
PY  - 2026
JO  - Bioscience reports
DO  - 10.1042/bsr20260182
UR  - https://doi.org/10.1042/bsr20260182
ER  -

APA

X, S., W, Z., C, C., H, Z., & Z, L. (2026). IGFBP2 attenuates high glucose-induced endothelial dysfunction through PI3K/Akt/eNOS pathway activation.. Bioscience reports. https://doi.org/10.1042/bsr20260182

Source records

pubmed · retrieved 2026-11-10T14:21:30.687Z