Cancer-associated SF3B1 mutation K700E causes widespread changes in U2/branchpoint recognition without altering splicing.

Damianov A, Lin CH, Zhang J, Manley JL, Black DL

Open source

DOI
10.1073/pnas.2423776122
Published
2025 Apr
Container
Proceedings of the National Academy of Sciences of the United States of America
Publisher
Not recorded
Open access
yes

Credibility signals

limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1073/pnas.2423776122,
  title = {Cancer-associated SF3B1 mutation K700E causes widespread changes in U2/branchpoint recognition without altering splicing.},
  author = {Damianov A and Lin CH and Zhang J and Manley JL and Black DL},
  year = {2025},
  journal = {Proceedings of the National Academy of Sciences of the United States of America},
  doi = {10.1073/pnas.2423776122},
  url = {https://doi.org/10.1073/pnas.2423776122}
}

RIS

TY  - JOUR
TI  - Cancer-associated SF3B1 mutation K700E causes widespread changes in U2/branchpoint recognition without altering splicing.
AU  - Damianov A
AU  - Lin CH
AU  - Zhang J
AU  - Manley JL
AU  - Black DL
PY  - 2025
JO  - Proceedings of the National Academy of Sciences of the United States of America
DO  - 10.1073/pnas.2423776122
UR  - https://doi.org/10.1073/pnas.2423776122
ER  - 

APA

A, D., CH, L., J, Z., JL, M., & DL, B. (2025). Cancer-associated SF3B1 mutation K700E causes widespread changes in U2/branchpoint recognition without altering splicing.. Proceedings of the National Academy of Sciences of the United States of America. https://doi.org/10.1073/pnas.2423776122

Source records