Insulin stimulates PKCzeta -mediated phosphorylation of insulin receptor substrate-1 (IRS-1). A self-attenuated mechanism to negatively regulate the function of IRS proteins.
- DOI
- 10.1074/jbc.m007281200
- Published
- 2001 Apr 27
- Container
- The Journal of biological chemistry
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1074/jbc.m007281200,
title = {Insulin stimulates PKCzeta -mediated phosphorylation of insulin receptor substrate-1 (IRS-1). A self-attenuated mechanism to negatively regulate the function of IRS proteins.},
author = {Liu YF and Paz K and Herschkovitz A and Alt A and Tennenbaum T and Sampson SR and Ohba M and Kuroki T and LeRoith D and Zick Y},
year = {2001},
journal = {The Journal of biological chemistry},
doi = {10.1074/jbc.m007281200},
url = {https://doi.org/10.1074/jbc.m007281200}
}RIS
TY - JOUR TI - Insulin stimulates PKCzeta -mediated phosphorylation of insulin receptor substrate-1 (IRS-1). A self-attenuated mechanism to negatively regulate the function of IRS proteins. AU - Liu YF AU - Paz K AU - Herschkovitz A AU - Alt A AU - Tennenbaum T AU - Sampson SR AU - Ohba M AU - Kuroki T AU - LeRoith D AU - Zick Y PY - 2001 JO - The Journal of biological chemistry DO - 10.1074/jbc.m007281200 UR - https://doi.org/10.1074/jbc.m007281200 ER -
APA
YF, L., K, P., A, H., A, A., T, T., SR, S., M, O., T, K., D, L., & Y, Z. (2001). Insulin stimulates PKCzeta -mediated phosphorylation of insulin receptor substrate-1 (IRS-1). A self-attenuated mechanism to negatively regulate the function of IRS proteins.. The Journal of biological chemistry. https://doi.org/10.1074/jbc.m007281200
Source records
- pubmed · retrieved 2026-09-24T23:39:02.105Z