Depletion of Pleckstrin Homology Domain Leucine-rich Repeat Protein Phosphatases 1 and 2 by Bcr-Abl Promotes Chronic Myelogenous Leukemia Cell Proliferation through Continuous Phosphorylation of Akt Isoforms

Isao Hirano, Satoki Nakamura, Daisuke Yokota, Takaaki Ono, Kazuyuki Shigeno, Shinya Fujisawa, Kaori Shinjo, Kazunori Ohnishi

Open source

DOI
10.1074/jbc.m808182200
Published
2009-08
Container
Journal of Biological Chemistry
Publisher
Elsevier BV
Open access
unknown

Credibility signals

serious concern Score 29/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1074/jbc.m808182200,
  title = {Depletion of Pleckstrin Homology Domain Leucine-rich Repeat Protein Phosphatases 1 and 2 by Bcr-Abl Promotes Chronic Myelogenous Leukemia Cell Proliferation through Continuous Phosphorylation of Akt Isoforms},
  author = {Isao Hirano and Satoki Nakamura and Daisuke Yokota and Takaaki Ono and Kazuyuki Shigeno and Shinya Fujisawa and Kaori Shinjo and Kazunori Ohnishi},
  year = {2009},
  journal = {Journal of Biological Chemistry},
  doi = {10.1074/jbc.m808182200},
  url = {https://doi.org/10.1074/jbc.m808182200}
}

RIS

TY  - JOUR
TI  - Depletion of Pleckstrin Homology Domain Leucine-rich Repeat Protein Phosphatases 1 and 2 by Bcr-Abl Promotes Chronic Myelogenous Leukemia Cell Proliferation through Continuous Phosphorylation of Akt Isoforms
AU  - Isao Hirano
AU  - Satoki Nakamura
AU  - Daisuke Yokota
AU  - Takaaki Ono
AU  - Kazuyuki Shigeno
AU  - Shinya Fujisawa
AU  - Kaori Shinjo
AU  - Kazunori Ohnishi
PY  - 2009
JO  - Journal of Biological Chemistry
DO  - 10.1074/jbc.m808182200
UR  - https://doi.org/10.1074/jbc.m808182200
ER  - 

APA

Hirano, I., Nakamura, S., Yokota, D., Ono, T., Shigeno, K., Fujisawa, S., Shinjo, K., & Ohnishi, K. (2009). Depletion of Pleckstrin Homology Domain Leucine-rich Repeat Protein Phosphatases 1 and 2 by Bcr-Abl Promotes Chronic Myelogenous Leukemia Cell Proliferation through Continuous Phosphorylation of Akt Isoforms. Journal of Biological Chemistry. https://doi.org/10.1074/jbc.m808182200

Source records