GJB2, a novel transcription target of HSF4, confers tumorigenic and metastatic phenotypes and sustains mitochondrial homeostasis in lung adenocarcinoma via the PI3K/AKT pathway
- DOI
- 10.1080/19336918.2026.2728214
- Published
- 2026-09-06
- Container
- Cell Adhesion & Migration
- Publisher
- Informa UK Limited
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1080/19336918.2026.2728214,
title = {GJB2, a novel transcription target of HSF4, confers tumorigenic and metastatic phenotypes and sustains mitochondrial homeostasis in lung adenocarcinoma via the PI3K/AKT pathway},
author = {Qing Hu and Renfan Zhang and Yefeng Zhou and Yang Yang and Hao Zhou and Xin Han and Shengrong Lin},
year = {2026},
journal = {Cell Adhesion \& Migration},
doi = {10.1080/19336918.2026.2728214},
url = {https://doi.org/10.1080/19336918.2026.2728214}
}RIS
TY - JOUR TI - GJB2, a novel transcription target of HSF4, confers tumorigenic and metastatic phenotypes and sustains mitochondrial homeostasis in lung adenocarcinoma via the PI3K/AKT pathway AU - Qing Hu AU - Renfan Zhang AU - Yefeng Zhou AU - Yang Yang AU - Hao Zhou AU - Xin Han AU - Shengrong Lin PY - 2026 JO - Cell Adhesion & Migration DO - 10.1080/19336918.2026.2728214 UR - https://doi.org/10.1080/19336918.2026.2728214 ER -
APA
Hu, Q., Zhang, R., Zhou, Y., Yang, Y., Zhou, H., Han, X., & Lin, S. (2026). GJB2, a novel transcription target of HSF4, confers tumorigenic and metastatic phenotypes and sustains mitochondrial homeostasis in lung adenocarcinoma via the PI3K/AKT pathway. Cell Adhesion & Migration. https://doi.org/10.1080/19336918.2026.2728214
Source records
- crossref · retrieved 2026-09-26T08:16:14.125Z