Ras-AKT signaling represses the phosphorylation of histone H1.5 at threonine 10 via GSK3 to promote the progression of glioma.

Sang B, Sun J, Yang D, Xu Z, Wei Y

Open source

DOI
10.1080/21691401.2019.1638795
Published
2019 Dec
Container
Artificial cells, nanomedicine, and biotechnology
Publisher
Not recorded
Open access
unknown

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BibTeX

@article{allodium:10.1080/21691401.2019.1638795,
  title = {Ras-AKT signaling represses the phosphorylation of histone H1.5 at threonine 10 via GSK3 to promote the progression of glioma.},
  author = {Sang B and Sun J and Yang D and Xu Z and Wei Y},
  year = {2019},
  journal = {Artificial cells, nanomedicine, and biotechnology},
  doi = {10.1080/21691401.2019.1638795},
  url = {https://doi.org/10.1080/21691401.2019.1638795}
}

RIS

TY  - JOUR
TI  - Ras-AKT signaling represses the phosphorylation of histone H1.5 at threonine 10 via GSK3 to promote the progression of glioma.
AU  - Sang B
AU  - Sun J
AU  - Yang D
AU  - Xu Z
AU  - Wei Y
PY  - 2019
JO  - Artificial cells, nanomedicine, and biotechnology
DO  - 10.1080/21691401.2019.1638795
UR  - https://doi.org/10.1080/21691401.2019.1638795
ER  - 

APA

B, S., J, S., D, Y., Z, X., & Y, W. (2019). Ras-AKT signaling represses the phosphorylation of histone H1.5 at threonine 10 via GSK3 to promote the progression of glioma.. Artificial cells, nanomedicine, and biotechnology. https://doi.org/10.1080/21691401.2019.1638795

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