Activation of lncRNA lnc-SLC4A1-1 induced by H3K27 acetylation promotes the development of breast cancer via activating CXCL8 and NF-kB pathway.

Yi T, Zhou X, Sang K, Huang X, Zhou J, Ge L

Open source

DOI
10.1080/21691401.2019.1664559
Published
2019 Dec
Container
Artificial cells, nanomedicine, and biotechnology
Publisher
Not recorded
Open access
unknown

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BibTeX

@article{allodium:10.1080/21691401.2019.1664559,
  title = {Activation of lncRNA lnc-SLC4A1-1 induced by H3K27 acetylation promotes the development of breast cancer via activating CXCL8 and NF-kB pathway.},
  author = {Yi T and Zhou X and Sang K and Huang X and Zhou J and Ge L},
  year = {2019},
  journal = {Artificial cells, nanomedicine, and biotechnology},
  doi = {10.1080/21691401.2019.1664559},
  url = {https://doi.org/10.1080/21691401.2019.1664559}
}

RIS

TY  - JOUR
TI  - Activation of lncRNA lnc-SLC4A1-1 induced by H3K27 acetylation promotes the development of breast cancer via activating CXCL8 and NF-kB pathway.
AU  - Yi T
AU  - Zhou X
AU  - Sang K
AU  - Huang X
AU  - Zhou J
AU  - Ge L
PY  - 2019
JO  - Artificial cells, nanomedicine, and biotechnology
DO  - 10.1080/21691401.2019.1664559
UR  - https://doi.org/10.1080/21691401.2019.1664559
ER  - 

APA

T, Y., X, Z., K, S., X, H., J, Z., & L, G. (2019). Activation of lncRNA lnc-SLC4A1-1 induced by H3K27 acetylation promotes the development of breast cancer via activating CXCL8 and NF-kB pathway.. Artificial cells, nanomedicine, and biotechnology. https://doi.org/10.1080/21691401.2019.1664559

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