Overexpression of HO-1 Contributes to Sepsis-Induced Immunosuppression by Modulating the Th1/Th2 Balance and Regulatory T-Cell Function.

Yoon SJ, Kim SJ, Lee SM.

DOI: 10.1093/infdis/jix142
Published: 2017-05-01
Container: J Infect Dis
Publisher: Not recorded
Open access: no

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Cite this work

BibTeX

@article{allodium:10.1093/infdis/jix142,
  title = {Overexpression of HO-1 Contributes to Sepsis-Induced Immunosuppression by Modulating the Th1/Th2 Balance and Regulatory T-Cell Function.},
  author = {Yoon SJ and  Kim SJ and  Lee SM.},
  year = {2017},
  journal = {J Infect Dis},
  doi = {10.1093/infdis/jix142},
  url = {https://doi.org/10.1093/infdis/jix142}
}

RIS

TY  - JOUR
TI  - Overexpression of HO-1 Contributes to Sepsis-Induced Immunosuppression by Modulating the Th1/Th2 Balance and Regulatory T-Cell Function.
AU  - Yoon SJ
AU  -  Kim SJ
AU  -  Lee SM.
PY  - 2017
JO  - J Infect Dis
DO  - 10.1093/infdis/jix142
UR  - https://doi.org/10.1093/infdis/jix142
ER  -

APA

SJ, Y., SJ, K., & SM., L. (2017). Overexpression of HO-1 Contributes to Sepsis-Induced Immunosuppression by Modulating the Th1/Th2 Balance and Regulatory T-Cell Function.. J Infect Dis. https://doi.org/10.1093/infdis/jix142

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europe-pmc · retrieved 2026-09-24T19:26:05.738Z