Elevated urea levels in human frontotemporal dementia and amyotrophic lateral sclerosis post-mortem brain tissue: evidence of a multi-dementia pathogenic mechanism.

Philbert SA, Church SJ, Unwin RD, Cooper GJS

Open source

DOI
10.1093/molecular-omics/aaiag019
Published
2026 Jul 14
Container
Molecular omics
Publisher
Not recorded
Open access
no

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BibTeX

@article{allodium:10.1093/molecular-omics/aaiag019,
  title = {Elevated urea levels in human frontotemporal dementia and amyotrophic lateral sclerosis post-mortem brain tissue: evidence of a multi-dementia pathogenic mechanism.},
  author = {Philbert SA and Church SJ and Unwin RD and Cooper GJS},
  year = {2026},
  journal = {Molecular omics},
  doi = {10.1093/molecular-omics/aaiag019},
  url = {https://doi.org/10.1093/molecular-omics/aaiag019}
}

RIS

TY  - JOUR
TI  - Elevated urea levels in human frontotemporal dementia and amyotrophic lateral sclerosis post-mortem brain tissue: evidence of a multi-dementia pathogenic mechanism.
AU  - Philbert SA
AU  - Church SJ
AU  - Unwin RD
AU  - Cooper GJS
PY  - 2026
JO  - Molecular omics
DO  - 10.1093/molecular-omics/aaiag019
UR  - https://doi.org/10.1093/molecular-omics/aaiag019
ER  - 

APA

SA, P., SJ, C., RD, U., & GJS, C. (2026). Elevated urea levels in human frontotemporal dementia and amyotrophic lateral sclerosis post-mortem brain tissue: evidence of a multi-dementia pathogenic mechanism.. Molecular omics. https://doi.org/10.1093/molecular-omics/aaiag019

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