ITCH-Mediated Ubiquitination and Degradation of THBS1: A Key Mechanism for Enhancing Mitochondrial Biogenesis and Alleviating Mouse Skeletal Muscle Atrophy.
- DOI
- 10.1111/apha.70299
- Published
- 2026 Oct
- Container
- Acta physiologica (Oxford, England)
- Publisher
- Not recorded
- Open access
- yes
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limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.
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Cite this work
BibTeX
@article{allodium:10.1111/apha.70299,
title = {ITCH-Mediated Ubiquitination and Degradation of THBS1: A Key Mechanism for Enhancing Mitochondrial Biogenesis and Alleviating Mouse Skeletal Muscle Atrophy.},
author = {Yu W and Wang Y and Li N and Wang D},
year = {2026},
journal = {Acta physiologica (Oxford, England)},
doi = {10.1111/apha.70299},
url = {https://doi.org/10.1111/apha.70299}
}RIS
TY - JOUR TI - ITCH-Mediated Ubiquitination and Degradation of THBS1: A Key Mechanism for Enhancing Mitochondrial Biogenesis and Alleviating Mouse Skeletal Muscle Atrophy. AU - Yu W AU - Wang Y AU - Li N AU - Wang D PY - 2026 JO - Acta physiologica (Oxford, England) DO - 10.1111/apha.70299 UR - https://doi.org/10.1111/apha.70299 ER -
APA
W, Y., Y, W., N, L., & D, W. (2026). ITCH-Mediated Ubiquitination and Degradation of THBS1: A Key Mechanism for Enhancing Mitochondrial Biogenesis and Alleviating Mouse Skeletal Muscle Atrophy.. Acta physiologica (Oxford, England). https://doi.org/10.1111/apha.70299
Source records
- pubmed · retrieved 2026-09-25T04:48:24.058Z