A reduction state potentiates the glucocorticoid response through receptor protein stabilization.

Kitagawa H, Yamaoka I, Akimoto C, Kase I, Mezaki Y, Shimizu T, Kato S

DOI: 10.1111/j.1365-2443.2007.01131.x
Published: 2007 Nov
Container: Genes to cells : devoted to molecular & cellular mechanisms
Publisher: Not recorded
Open access: unknown

Credibility signals

serious concern Score 8/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

cautionDOI registered: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
cautionDOI resolves: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
serious concernRetraction Watch retraction: 1 retraction notice matched this DOI. Source: Retraction Watch; license: CC BY 4.0
not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
not scoredRetraction Watch correction: No correction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
not scoredOpen access status: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer

Cite this work

BibTeX

@article{allodium:10.1111/j.1365-2443.2007.01131.x,
  title = {A reduction state potentiates the glucocorticoid response through receptor protein stabilization.},
  author = {Kitagawa H and Yamaoka I and Akimoto C and Kase I and Mezaki Y and Shimizu T and Kato S},
  year = {2007},
  journal = {Genes to cells : devoted to molecular \& cellular mechanisms},
  doi = {10.1111/j.1365-2443.2007.01131.x},
  url = {https://doi.org/10.1111/j.1365-2443.2007.01131.x}
}

RIS

TY  - JOUR
TI  - A reduction state potentiates the glucocorticoid response through receptor protein stabilization.
AU  - Kitagawa H
AU  - Yamaoka I
AU  - Akimoto C
AU  - Kase I
AU  - Mezaki Y
AU  - Shimizu T
AU  - Kato S
PY  - 2007
JO  - Genes to cells : devoted to molecular & cellular mechanisms
DO  - 10.1111/j.1365-2443.2007.01131.x
UR  - https://doi.org/10.1111/j.1365-2443.2007.01131.x
ER  -

APA

H, K., I, Y., C, A., I, K., Y, M., T, S., & S, K. (2007). A reduction state potentiates the glucocorticoid response through receptor protein stabilization.. Genes to cells : devoted to molecular & cellular mechanisms. https://doi.org/10.1111/j.1365-2443.2007.01131.x

Source records

pubmed · retrieved 2026-09-25T14:59:41.976Z