Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws.
- DOI
- 10.1111/jdi.12048
- Published
- 2013 Jan 29
- Container
- Journal of diabetes investigation
- Publisher
- Not recorded
- Open access
- yes
Credibility signals
limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.
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Cite this work
BibTeX
@article{allodium:10.1111/jdi.12048,
title = {Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws.},
author = {Lee HK and Shim EB},
year = {2013},
journal = {Journal of diabetes investigation},
doi = {10.1111/jdi.12048},
url = {https://doi.org/10.1111/jdi.12048}
}RIS
TY - JOUR TI - Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws. AU - Lee HK AU - Shim EB PY - 2013 JO - Journal of diabetes investigation DO - 10.1111/jdi.12048 UR - https://doi.org/10.1111/jdi.12048 ER -
APA
HK, L., & EB, S. (2013). Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws.. Journal of diabetes investigation. https://doi.org/10.1111/jdi.12048
Source records
- pubmed · retrieved 2026-09-25T14:32:10.580Z