FGF23 promotes atrial fibrillation susceptibility through ETV1-dependent sodium channel regulation and calcium handling abnormalities.
- DOI
- 10.1113/ep093891
- Published
- 2026 Aug 24
- Container
- Experimental physiology
- Publisher
- Not recorded
- Open access
- yes
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limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.
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Cite this work
BibTeX
@article{allodium:10.1113/ep093891,
title = {FGF23 promotes atrial fibrillation susceptibility through ETV1-dependent sodium channel regulation and calcium handling abnormalities.},
author = {Yu Q and Zeng L and Chen Z and Lin Z and Wang C and Wang L and Fu H and Zhang J},
year = {2026},
journal = {Experimental physiology},
doi = {10.1113/ep093891},
url = {https://doi.org/10.1113/ep093891}
}RIS
TY - JOUR TI - FGF23 promotes atrial fibrillation susceptibility through ETV1-dependent sodium channel regulation and calcium handling abnormalities. AU - Yu Q AU - Zeng L AU - Chen Z AU - Lin Z AU - Wang C AU - Wang L AU - Fu H AU - Zhang J PY - 2026 JO - Experimental physiology DO - 10.1113/ep093891 UR - https://doi.org/10.1113/ep093891 ER -
APA
Q, Y., L, Z., Z, C., Z, L., C, W., L, W., H, F., & J, Z. (2026). FGF23 promotes atrial fibrillation susceptibility through ETV1-dependent sodium channel regulation and calcium handling abnormalities.. Experimental physiology. https://doi.org/10.1113/ep093891
Source records
- pubmed · retrieved 2026-09-24T23:05:36.512Z
- europe-pmc · retrieved 2026-09-24T23:05:36.485Z