Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells.

Takada Y, Sethi G, Sung B, Aggarwal BB

Open source

DOI
10.1124/mol.107.041350
Published
2008 May
Container
Molecular pharmacology
Publisher
Not recorded
Open access
unknown

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BibTeX

@article{allodium:10.1124/mol.107.041350,
  title = {Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells.},
  author = {Takada Y and Sethi G and Sung B and Aggarwal BB},
  year = {2008},
  journal = {Molecular pharmacology},
  doi = {10.1124/mol.107.041350},
  url = {https://doi.org/10.1124/mol.107.041350}
}

RIS

TY  - JOUR
TI  - Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells.
AU  - Takada Y
AU  - Sethi G
AU  - Sung B
AU  - Aggarwal BB
PY  - 2008
JO  - Molecular pharmacology
DO  - 10.1124/mol.107.041350
UR  - https://doi.org/10.1124/mol.107.041350
ER  - 

APA

Y, T., G, S., B, S., & BB, A. (2008). Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells.. Molecular pharmacology. https://doi.org/10.1124/mol.107.041350

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